Figure 5From: Immunological hotspots analyzed by docking simulations: evidence for a general mechanism in pemphigus vulgaris pathology and transformationRedundancy profile of predicted Dsg1 peptides to Dsg3 sequences. The number of predicted high-affinity Dsg1 (15 mer) peptide sequences shown as a function of the number of substitutions from the highest identity peptide sequence derived from the Dsg3 proteome derived using a 15 mer sliding window.Back to article page