Figure 4
From: Aquaporin-4 autoantibodies increase vasogenic edema formation and infarct size in a rat stroke model
Edema formation within the basal ganglia and the cortex. T2-relaxation time, a parameter that correlates closely with vasogenic edema formation, was comparable between NMO-IgG and control-IgG treated animals if measured within the basal ganglia (p > 0.05). Vasogenic edema formation within the cortex was significantly increased in animals treated with NMO-IgG as compared to control-IgG (p < 0.05).