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Fig. 1 | BMC Immunology

Fig. 1

From: Platelet-derived extracellular vesicles promote endothelial dysfunction in sepsis by enhancing neutrophil extracellular traps

Fig. 1

Characterization of platelet-derived extracellular vesicles (PEVs) in sepsis patients and healthy volunteers. A: The morphology of extracellular vesicles (EVs) isolated from clinical blood samples was observed by transmission electron microscopy (TEM); B: The diameter of EVs isolated from clinical blood samples was analyzed by nanoparticle tracking analysis (NTA); C: The proportion of EVs with surface markers CD41 + or CD61 + in isolated EVs was detected by flow cytometry. D: Comparison of the number of PEVs (CD41 + EVs) in clinical blood samples from normal healthy volunteers and patients with sepsis, **P < 0.01 vs. Normal group. Normal (n = 30) and Sepsis (n = 30) group, EV: extracellular vesicle; TEM, transmission electron microscopy; NTA, nanoparticle tracking analysis

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