Fig. 3From: Platelet-derived extracellular vesicles promote endothelial dysfunction in sepsis by enhancing neutrophil extracellular trapsCorrelation of the number of PEVs with markers of NET formation and endothelial injury. A-D: Linear regression assessed the correlation between the number of PEVs and histone H3 (A), MPO (B), angiopoietin-2 (C) and endocan (D) level in sepsis patients (n = 30). PEV, platelet-derived extracellular vesicles; MPO, myeloperoxidaseBack to article page