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Fig. 5 | BMC Immunology

Fig. 5

From: Platelet-derived extracellular vesicles promote endothelial dysfunction in sepsis by enhancing neutrophil extracellular traps

Fig. 5

PEVs promote NET formation and endothelial function impairment in sepsis rats. A-F: The level of markers of NET formation: histone H3 (A), HMGB1 (B) and MPO (C), as well as level of markers of endothelial function impairment: angiopoietin-2 (D), endocan (E) and syndecan-1 (F) in the rats’ blood in each group (n = 8), were measured by ELISA, *P < 0.05 and **P < 0.01 vs. Sham group, #P < 0.05 and ##P < 0.01 vs. CLP group. NET, neutrophil extracellular traps; HMGB1, high mobility group protein B1; MPO, myeloperoxidase; CLP, cecal ligation and puncture

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BMC Immunology

ISSN: 1471-2172

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